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This page (revision-7) was last changed on 26-Dec-2012 11:38 by 1614216108

This page was created on 16-Apr-2012 12:03 by UnknownAuthor

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At line 3 changed 2 lines
中文名称：胆囊收缩素-促胰酶素\\
英文名称：cholecystokinin-pancerozymin,CCK-PZ,CCK\\
　　中文名称：胆囊收缩素-促胰酶素\\
　　英文名称：cholecystokinin-pancerozymin,CCK-PZ,CCK\\
At line 10 changed 4 lines
　　1.急性胰腺炎发病，胰腺外分泌减低，十二指肠内胰酶活性特别是胰蛋白酶活性减低。由于负反馈作用CCK分泌增多。CCK是胰腺炎加重的因素，有望开发CCK拮抗药物或CCK受体阻断药物治疗胰腺炎。\\
　　2.慢性胰腺炎CCK血浓度较正常人高，进餐后升高更明显。有痛性慢性胰腺炎考虑有可能是由于增高的CCK的持续刺激作用，测定CCK为不可缺少。CCK受体阻断剂对缓解慢性胰腺炎的疼痛有效。\\
　　3.阻塞性黄疸血CCK升高。由于十二指肠内缺乏胆汁，胰蛋白酶原活化降低，引起CCK分泌亢进。\\
　　4.肝硬化，由于CCK在肝脏代谢障碍，血浓度升高约是正常人的3倍。\\
1.急性胰腺炎发病，胰腺外分泌减低，十二指肠内胰酶活性特别是胰蛋白酶活性减低。由于负反馈作用CCK分泌增多。CCK是胰腺炎加重的因素，有望开发CCK拮抗药物或CCK受体阻断药物治疗胰腺炎。\\
2.慢性胰腺炎CCK血浓度较正常人高，进餐后升高更明显。有痛性慢性胰腺炎考虑有可能是由于增高的CCK的持续刺激作用，测定CCK为不可缺少。CCK受体阻断剂对缓解慢性胰腺炎的疼痛有效。\\
3.阻塞性黄疸血CCK升高。由于十二指肠内缺乏胆汁，胰蛋白酶原活化降低，引起CCK分泌亢进。\\
4.肝硬化，由于CCK在肝脏代谢障碍，血浓度升高约是正常人的3倍。\\
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空腹10-12个小时以上。\\
标本采集与运送\\
EDTA或肝素抗凝，加抑肽酶500U/1ml抑制分解，静脉采血，30分钟内分离血浆，零下20摄氏度以下保存。\\
　　空腹10-12个小时以上。\\
At line 24 added 2 lines
%%tab-标本采集和运送要求
　　EDTA或肝素抗凝，加抑肽酶500U/1ml抑制分解，静脉采血，30分钟内分离血浆，零下20摄氏度以下保存。\\
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Version Date Modified Size Author Changes ... Change note
7 26-Dec-2012 11:38 1.034 kB 1614216108 to previous
6 26-Dec-2012 11:06 1.013 kB 1614216108 to previous | to last 胃肠胰激素 ==> 血.胃肠胰激素
5 05-Dec-2012 17:10 1.011 kB 1614216108 to previous | to last 胆囊收缩素-促胰酶素 ==> 胆囊收缩素-促胰酶素测定
4 05-Dec-2012 17:10 1.011 kB 1614216108 to previous | to last
3 04-May-2012 09:32 1.022 kB 1614216108 to previous | to last
2 23-Apr-2012 10:40 1.0 kB 1614216108 to previous | to last
1 16-Apr-2012 12:03 0.99 kB UnknownAuthor to last
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